Sebastian D. Amigorena - Nanotechnology

Author Information

Sébastian Amigorena is a renowned researcher affiliated with INSERM U932 at the Institute Curie, located in Paris, France. His work primarily focuses on immunology and cancer research, contributing significantly to our understanding of immune responses and tumor biology. His affiliation with such a prestigious institution underlines his position as a leading figure in his field.

Research Contributions

Sébastian Amigorena has made substantial contributions to the fields of immunology and cancer research. His recent work includes studies on plasma cell maintenance, antibody secretion, and the mechanisms of immune cell responses. His research often employs advanced techniques such as genome-wide CRISPR screens to uncover intrinsic checkpoints in immune responses. Additionally, his work on epigenetics and the tumor microenvironment has provided valuable insights into T cell differentiation and the dynamics of tumor-infiltrating immune cells.

Aliases

Sébastian Amigorena is known by various aliases in academic publications, including S. Amigorena, Sébastian Amigorena, Sebastián Amigorena, Sebastian D. Amigorena, Sebastian Amigoréna, and other variations. These aliases reflect different transliterations and spellings of his name found in research databases and publications.

Publication and Citation Metrics

Metric	Value
Total Citation Count	49,119
h-index	93
Total Paper Count	278

Publications:

Plasma cell maintenance and antibody secretion are under the control of Sec22b-mediated regulation of organelle dynamics

DOI: 10.1101/2022.01.14.476154

Year: 2022

Splicing patterns in SF3B1 mutated uveal melanoma generate shared immunogenic tumor-specific neo-epitopes.

DOI: 10.1158/2159-8290.CD-20-0555

Year: 2021

In vivo genome-wide CRISPR screens identify SOCS1 as a major intrinsic checkpoint of CD4+ Th1 cell response

DOI: 10.1101/2021.04.12.439455

Year: 2021

Publisher Correction: The receptor DNGR-1 signals for phagosomal rupture to promote cross-presentation of dead-cell-associated antigens

DOI: 10.1038/s41590-021-00885-6

Year: 2021

Induction of transposable element expression is central to innate sensing

DOI: 10.1101/2021.09.10.457789

Year: 2021

Contribution of resident and circulating precursors to tumor-infiltrating CD8+ T cell populations in lung cancer

DOI: 10.1126/sciimmunol.abd5778

Year: 2021

In vivo genome-wide CRISPR screens identify SOCS1 as intrinsic checkpoint of CD4+ TH1 cell response

DOI: 10.1126/sciimmunol.abe8219

Year: 2021

Alternative Enhancer Usage and Targeted Polycomb Marking Hallmark Promoter Choice during T Cell Differentiation.

DOI: 10.1016/j.celrep.2020.108048

Year: 2020

Epigenetics of T cell fate decision.

DOI: 10.1016/j.coi.2020.01.002

Year: 2020

Tumor invasion in draining lymph nodes is associated with Treg accumulation in breast cancer patients

DOI: 10.1038/s41467-020-17046-2

Year: 2020

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