Gerald R. Crabtree - Nanotechnology

Author Information

Gerald R. Crabtree is a prominent researcher affiliated with the Department of Pathology at Stanford University School of Medicine, located in Stanford, CA, USA. His work is primarily focused on the field of molecular biology and genetics, with significant contributions to understanding chromatin remodeling and its implications in various diseases, including cancer and neurological disorders.

Research Contributions

Gerald R. Crabtree has made substantial contributions to the field of molecular biology, with a particular focus on chromatin remodeling complexes such as mSWI/SNF. His research explores the role of these complexes in gene regulation, cancer, and neurological disorders. Notable studies include investigations into the mechanisms of oncogenic transformation in gastric cancer using CRISPR/Cas9-engineered models and the impact of serotonin signaling on social deficits in autism spectrum disorders. His work also delves into the regulation of Polycomb Repressive Complex and HOX genes, providing critical insights into genetic and epigenetic regulation.

Aliases

Gerald R. Crabtree is also known by several aliases, including G Crabtree, G. R. Crabtree, Gerald Crabtree, and G R Crabtree. These variations are commonly found in academic publications and citations.

Publication and Citation Metrics

Metric	Value
Citation Count	57,129
h-index	119
Paper Count	347

Publications:

ACTL6a coordinates axonal caliber recognition and myelination in the peripheral nerve

DOI: 10.1016/j.isci.2022.104132

Year: 2022

mSWI/SNF interacts with the ribosome and its inhibition/mutations alter translation and sensitize to mTOR/PI3K inhibitors

DOI: 10.1101/2021.05.10.443459

Year: 2021

Sa134 A CRISPR/CAS9-ENGINEERED ARID1A-DEFICIENT HUMAN GASTRIC CANCER ORGANOID MODEL REVEALS ESSENTIAL AND NON-ESSENTIAL MODES OF ONCOGENIC TRANSFORMATION

DOI: 10.1016/S0016-5085(21)01747-9

Year: 2021

ARID1B is a Dosage-sensitive Regulator of Polycomb Repressive Complex Distribution and HOX Gene Regulation in Patient-derived Neural Progenitors

DOI: 10.21203/rs.3.rs-959800/v1

Year: 2021

Increased ACTL6A occupancy within mSWI/SNF chromatin remodelers drives human squamous cell carcinoma.

DOI: 10.1016/j.molcel.2021.10.005

Year: 2021

mSWI/SNF promotes Polycomb repression both directly and through genome-wide redistribution.

DOI: 10.1038/s41594-021-00604-7

Year: 2021

Systemic enhancement of serotonin signaling reverses social deficits in multiple mouse models for ASD

DOI: 10.1038/s41386-021-01091-6

Year: 2021

Increased ACTL6A Occupancy Within mSWI/SNF Chromatin Remodelers Drives Human Squamous Cell Carcinoma

DOI: 10.1101/2021.03.22.435873

Year: 2021

Increased ACTL6A Occupancy within mSWI/SNF Chromatin Remodelers Drives Human Squamous Cell Carcinoma

DOI: 10.2139/ssrn.3806798

Year: 2021

A CRISPR/Cas9-engineered ARID1A-deficient human gastric cancer organoid model reveals essential and non-essential modes of oncogenic transformation.

DOI: 10.1158/2159-8290.CD-20-1109

Year: 2021

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